Krishnaswamy A. (2016). Building connections. Science. Nov 4; 354(6312):558.

Krishnaswamy A.*, Yamagata M.*, Duan X., Sanes JR. (2015). Sidekick 2 directs formation of an object motion selective circuit. Nature. Aug 27;524(7566):466-70. * Equal contribution

Duan X.*, Krishnaswamy A.*, De La Huerta I., Sanes JR. (2014). Type II Cadherins Guide Assembly of a Direction-Selective Retinal Circuit. Cell. Aug 14;158(4):793-807* Equal contribution

Lilley BN., Krishnaswamy A., Wang Z., Kishi M., Frank E., Sanes JR. (2014). SAD kinases control the maturation of nerve terminals in the mammalian peripheral and central nervous systems. PNAS. Jan 21;111(3):1138-43.

Davisson MT., Bronson RT., Tadenev AL., Motley WW., Krishnaswamy A., Seburn KL., Burgess RW. (2011). A spontaneous mutation in contactin 1 in the mouse. PLoS One;6(12).

Krishnaswamy A., Cooper E. (2011) Reactive Oxygen Species Inactivate Neuronal Nicotinic Acetylcholine Receptors Through a Highly Conserved Cysteine Near the Intracellular Mouth of the Channel: Implications for Diseases that Involve Oxidative Stress. J. Physiol. Jan 1;590(Pt 1):39-47.

Campanucci VA.*, Krishnaswamy A.*, Cooper E. (2010) Diabetes depresses synaptic transmission in sympathetic ganglia by inactivating nAChRs through a conserved intracellular cysteine residue.  Neuron. 66(6):827-34. * Equal contribution

Caffery, P.M.*, Krishnaswamy, A.*, Sanders, T., Hartlaub, H., Klysik, J., Cooper, E., Hawrot, E.(2009). Knock-in a -bungarotoxin sensitivity enables visualization and pharmacological characterization of postsynaptic a3- containing nicotinic acetylcholine receptors in a novel mouse model. Eur. J. Neurosci. Dec 3;30(11):2064-76. * Equal contribution

Krishnaswamy, A., Cooper E. (2009) An activity-dependent retrograde signal induces the expression of the high affinity choline transporter in cholinergic neurons. Neuron. 61:272-86.

Campanucci, V.A., Krishnaswamy, A., Cooper, E. (2008) Mitochondrial reactive oxygen species inactivate neuronal nicotinic acetylcholine receptors and induce long-term depression of fast nicotinic synaptic transmission. J Neurosci  28(7):1733-44.  

Rassadi, S.*, Krishnaswamy, A.*, Pié, B., McConnell, R., Jacob, M.H., Cooper E. (2005) A null mutation for the a3 nicotinic acetylcholine (ACh) receptor gene abolishes fast synaptic activity in sympathetic ganglia and reveals that ACh output from developing preganglionic terminals is regulated in an activity-dependent retrograde manner. J. Neurosci. 25(37):8555-66. * Equal contribution